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In contrast, integral membrane substrates with degrons present in TMDs or within cytoplasmic domains are recognized by HRD1 in an ER lectin-independent manner or by other ERAD E3 ubiquitin ligases equivalently or preferentially

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It also recruits inflammatory cells such as neutrophils, eosinophils, lymphocytes, and macrophages by potentiating the action of histamine, which injures lung tissues and causes inflammation [8, 9]

Here we report a germline heterozygous frame-shift mutation in the mismatch repair MLH1 gene which was identified in members of two unrelated LS families