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Description
Results We demonstrate a developmental perturbation in CCK-expressing excitatory neurons within the mPFC: their density increases sharply in early postnatal life, stabilizes during adolescence, and typically declines in adulthood in wild-type (WT) mice, yet remains persistently elevated in adult KI counterparts

ABCC10, ABCC11, and ABCC12

Over time, these can progress to more serious neurological abnormalities
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