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It is increasingly recognized that sex differences in pain could be associated with neuroimmune and glial mechanisms, which are key players in the neuroglial maladaptive plasticity that transitions acute pain to chronic pathological pain [31, 352356]
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PARylation Upon sensing DNA damage (e.g., single-strand breaks), PARPase (especially PARP1) is activated and rapidly accumulates at the site of damage
60 In hepatoma cells, miR-125b-5p directly targets the downstream gene STAT3 to enhance -catenin phosphorylation
