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Archived from the original on January 6, 2022

As these transcriptional alterations suggest a reduction in hepatic fibrosis in response to lowering oxalate via AGXT overexpression, we next evaluated hepatic fibrosis through histopathological and immunofluorescence analyses coupled with biochemical verification

Antioxidants Redox Signal (2018) 29(17):175673

This interaction was first suspected as the cause for the observed relapse and exacerbation of the neurological symptoms in pernicious anemia patients who were prescribed high oral doses of folic acid (46)