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MDA values and NO levels increased after the initial abdominal wall defect was made[36,37], and increase during subsequent adhesion formation[36,37]

We propose that scRNA-seq will reveal a distinct redox pseudotime trajectory within TILs, characterized by a significant covariance between mtROS stress signatures and markers of terminal exhaustion

Moreover, inhibition of Notch1 signaling also leads to accumulation of glutamine and increased activity of complex 1 in the mitochondrial electron transport chain, conferring resistance to treatment ( Figure 3 ) (72)
