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The positive correlation between GST gene polymorphisms and hepatocellular carcinoma caused by hepatitis B virus chronic infection [55] and uterine cancer associated to human papilloma virus infection was described [56] (Figure 3)
Leave-one-out analyses showed consistent effect estimates across all exclusions, with pooled risk ratios ranging from 0.59 to 0.77, and no single study reversing the direction of effect
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