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This suggests the hypothesis that the Semax/Cu 2+ complex mainly interacts with oligomeric species of A 140 by both retarding their formation and hampering their interaction with the lipidic membrane

[DOI] [PMC free article] [PubMed] [Google Scholar] 312.Willis AW, Evanoff BA, Lian M, Galarza A, Wegrzyn A, Schootman M, et al

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Research has demonstrated that inhibiting the opening of MPTP during short-term ischemia has limited protective effects on myocardial cells after reperfusion, but it plays a more significant role in protecting against reperfusion injury after long-term ischemia [71]