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When p53 was bound to FOXO4, p53 got stuck in the nucleus with the FOXO4 and never made it out the mitochondria where it could start apoptosis
Their findings suggested that NAC disrupts vWF multimers via disulfide bond reduction, reducing platelet aggregation and microthrombi formation, which are central to sepsis-induced organ failure (Liu et al., 2025)
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(2013), this modified form also inhibits the growth, development, and angiogenesis of experimental endometriosis in mice [192]
