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It promotes inflammation via NF-B pathway activation (82) and by activating the NLRP3 inflammasome in endothelial cells and arterial walls in mice, involving mitochondrial ROS, TXNIP, and lysosomal instability (101, 134)

Assim, impede a formao de nveis excessivos de triglicrides, normaliza o quadro lipoprotenico reduo das lipoprotenas de densidade muito baixa (VLDL) e o aumento das lipoprotenas de alta densidade (HDL) eliminando, um dos mais importantes fatores de risco de doenas cardiovasculares (aterosclerose e afeces cardacas consequentes

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120 Currently, research on D-Asp as a treatment for TRS disease is still in its early stages, and animal experiments are ongoing