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Silencing P65 or EAAT3 increases ROS levels and reduces GSH synthesis, but these changes alone are insufficient to trigger significant cell death due to the robust ferroptosis-resistance mechanisms in cancer cells, including the xCT-GSH-GPX4 system and other protective pathways

220.Toaha SM, Mollah BR, Ahammad MU

Some studies reported increased levels of catalase, whereas others reported downregulation of this enzyme

Subsequent research should concentrate on refined strategies for modulating lipid peroxidation in cancer therapy, encompassing the concurrent application of various antioxidants with standard treatments, evaluating oxidative stress profiles throughout therapy, and employing biomarkers to ascertain optimal timing and dosing of antioxidants