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When the cell suffers from damage, its membranes usually lose integrity and the intracellular LDH subsequently gets released outside [41]

As direct consequence, some SnCE1 acetylation sites are enzymatically deacetylated upon exposure in the host cell following injection of SnCE1 while others might remain stable as they are not accessible for enzymatic deacetylation

The metabolic intermediates produced as a result can support tumour cell survival by providing an alternate acetyl-CoA ester source for the TCA cycle

A detailed study revealed that polyphenols not only significantly increased intracellular ROS levels but also induced the upregulation of intracellular apoptotic stimulants, such as caspase-3, an important protease that plays a key role in apoptosis