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The investigation of the mechanism of glutathione deficiency in the lungs children with asthma has shown that dysregulation of glutathione-related antioxidant defense occurs due to dysfunction of a transcription factor that stimulates gene expression of the enzymes related to the antioxidant defense and glutathione production in cells

Despite its promising animal data, human studies on BPC 157 are sparse and not well-conducted, leaving significant uncertainty about its effects in humans

In: Ziegler E, Filer L, eds