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After being absorbed, B12 is gradually released from the sucrosome vesicles within the enterocytes and then transported into the bloodstream, leading to elevated circulatory B12 levels.This mechanism ensures that Sucrosomial B12 is absorbed efficiently, even in the presence of compromised IF function or damage to the ileum

While the in vitro model using KRAS-derived PDAC cells had a promising impact, the in vivo experiment, unfortunately, did not achieve reduced cell growth due to the adaptive metabolomic plasticity of those cells in response to CB-839 [43]

Understanding their differences helps clarify their potential applications