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Similar content being viewed by others Acknowledgements This work was supported by the Basic Science Research Program through the National Research Foundation of Korea (NRF) funded by the Ministry of Education (2020R1I1A3A04038150), Korea Basic Science Institute (National research Facilities and Equipment Center) grant funded by the Ministry of Education (2023R1A6C101A045), and the NRF grant funded by the Korea government (MSIT 2020R1A2C2103067)

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By incorporating miRNA structures into gene therapy agents, it aims to inhibit the production of the mutant Z-AAT protein and promote the production of functional A1AT ( These findings highlight the potential of A1AT augmentation therapy to modulate the inflammatory response in A1ATD, reduce neutrophil-mediated damage, and improve clinical outcomes in various manifestations of the disease, including lung inflammation and panniculitis