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Abstract The mechanistic (or mammalian) target of rapamycin (mTOR) and the adenosine monophosphate-activated protein kinase (AMPK) regulate cell survival and metabolism in response to diverse stimuli such as variations in amino acid content, changes in cellular bioenergetics, oxygen levels, neurotrophic factors and xenobiotics
To test whether the protective effect of ascites was specific to ferroptosis, we tested ascites impact on the cell viability of CAOV3 cells when treated with non-ferroptosis cell death inducers (e.g., staurosporine, actinomycin D, and rapamycin)
Temperature: maximum growth rates are attained by the most cyanobacteria above 25C
