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A speculative hypothesis is that the methylation status of the SNCA gene might be influenced by the gut microbiome
Here, we focus on ATP signaling, which is relevant to epithelial cells, and nitric oxide (NO) signaling, which tends to be perturbed in endothelial cells [70]
Consider intermittent fasting or caloric restriction, which may synergize with MOTS-c's AMPK-mediated effects
