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Reduced Fractalkine levels Lead to striatal synaptic plasticity deficits in Huntington's disease
Psychiatry 54 (10), 915922
Such alterations were also associated with inhibition of cytochrome c oxidase (CcOX) activity and microtubule network disassembly, which are considered hallmarks of nitric oxide (NO) toxicity, indicating that NO, rather than the depletion of GSH per se, is the primary mediator of cell damage
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