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The common sites of lipid ROS accumulation are the endoplasmic reticulum, mitochondria, and lysosomes (14, 20, 21)
Mice were housed in specific pathogen-free facilities at Johns Hopkins University and University of Alabama at Birmingham
These encompass the activation of iron-regulatory proteins, thereby elevating transferrin receptor expression, and the repression of the transcription factor CCAAT/enhancer-binding protein (C/EBP) or bone morphogenetic protein (BMP)-mediated Smad signaling pathways, resulting in attenuated hepcidin expression [779781]
doi: 10.1002/mds.25187
