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Although there were no electrophysiological correlates of the dystonic movements in the EEG, response to anti-seizure medication can suggest an underlying epileptic etiology
At the molecular level, it appears to interact with intracellular signaling systems essential for tissue adaptation
Inflammopharmacology 112
It also suppresses NF-kB activation and modulates macrophage polarization toward an M2 anti-inflammatory phenotype
