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Redox balance is maintained by prevention, interception, and repair, and concomitantly the regulatory potential of molecular thiol-driven master switches such as Nrf2/Keap1 or NF-B/IB is used for system-wide oxidative stress response [5]
That may be because GHK is present in various proteins that get broken down via hydrolysis during injury
All spectra were fit with multiple sub-spectra using Lorentzian lineshapes
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