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This metabolic dysregulation leads to an increased mobilization and subsequent influx of free fatty acids (FFAs) into the liver, thereby promoting the accumulation of TG and the development of hepatic steatosis

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doi: 10.1038/s43018-02300647-3 25 WangQWuMLiHRaoXAoLWangHet al

While anecdotal reports are positive, robust human clinical trials on NAD+ IV therapy are still limited