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Apoptosis is common in the testis as a safeguard mechanism for eliminating defective germ cells (Shukla et al., 2012) which is exacerbated by antidepressant usage inducing DNA fragmentation and lipid peroxidation, leading to cellular damage ( in vivo models is necessary due to testicular tissue complexity, these findings offer valuable insights into antidepressant-induced reproductive toxicity (Mytych et al., 2017)

In the immunopathological process of vitiligo, DCs and NK cells drive the differentiation of T cells into various effector subsets, such as Th1, Th17, and Treg cells, through multiple mechanisms (122)

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The BBB prevents almost 98% of small molecule drugs and nearly all large-molecule therapeutics such as recombinant peptides, proteins, anti-sense agents, and genetic vectors, from reaching the brain [1]