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Repression of BACH1 suppresses the spread of TNBC cells to the lungs and enhances their responsiveness to metformin, indicating that specifically blocking BACH1 can alter tumor metabolism by boosting mitochondrial metabolism
pentosus GSSK2 + HCD (Group V) demonstrated reduced hepatic inflammation and improved steatosis, with minimal fat deposition compared with HCD (Group II) animals (Fig
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Mol Plant-Microbe Interact 18:254259 Gapper NE, McQuinn RP, Giovaninni JJ (2013) Molecular and genetic regulation of fruit ripening
