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In vitro cell culture experiments supported these findings by showing increased fatty acid oxidation and upregulated expression of lipid-related genes
PPAR attenuates hepatic steatosis through autophagy-mediated fatty acid oxidation Peroxisome proliferator-activated receptor delta (PPAR), a member of the nuclear receptor family with implications in metabolic diseases, is found to significantly enhance hepatic autophagic flux
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noninferiority P = 0.020)
