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Description
In BM-MSCs, METTL14 regulated osteogenic differentiation via the mA/IGF2BPs/Beclin-1 signaling axis [126], while METTL16 suppressed osteogenesis by promoting mA modification of PPAR transcripts, thereby enhancing its expression [127]

ODonovan, James T

NDI1 expression also supported healthier adipose remodelling during HFD feeding, reflected by a shift towards smaller adipocytes with no difference in gWAT mass

Evidence suggests that metabolic substrates, such as lactate, may play a signaling role in synaptic modulation and plasticity (Mller et al., 2021)
