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Recently, a second cardiac K ATP channel complex has been identified at the surface of cardiomyocytes, consisting of a Kir6.1 pore-forming subunit that displays constitutive, rather than ATP-dependent, activity (Brennan et al., 2024)

Furthermore, elevated uric acid concentrations place additional stress on the kidneys, and prolonged hyperuricemia not only increases the risk of kidney disease but can also lead to kidney stones or even kidney failure (Fathallah-Shaykh & Cramer, 2014)

Other studies found that TMAO induces hypercholesterolemia by promoting intracellular CHO accumulationenhancing macrophage cholesterol accumulation via microbiota-dependent pathways involving the increased expression of pro-atherogenic scavenger receptor proteins CD36 and SRA on the cell surface (Wang et al., 2011)

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