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As with the majority of anticancer drugs, its cytotoxic action comes along with possible damage for different non-neoplastic tissues, inducing toxicities that may affect patients quality of life

In T2DM, elevated LC3-II and p62 levels suggest that autophagic flux is active but may be dysfunctional due to inefficient degradation of the autophagosomes

Recently, the presence of nuclear-encoded transcription factors (MOF, AP1, CEBPB, MEF2D), and the contribution of nuclear spliceosome complex to mtDNA-splicing provided strong evidence of control of mitochondrial replication/transcription demonstrating evolutionary adaptation of the mammalian mtDNA in the nucleoid [75]

[DOI] [PubMed] [Google Scholar] 315.Jacks T., Smith R., Judith F., Schleim K., Frazier E., Chen H., Krupa D., Hora D., Jr., Nargund R., Patchett A., et al