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Studies suggest that latent KSHV infection also upregulates glutamine metabolism, inducing protein expression of the glutamine transporter SLC1A5 (ASCT2) as well as MondoA and its downstream targets involved in regulation of glutaminolysis and making the infected cells reliant on glutaminolysis for survival [133]

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Erickson HK, Lewis Phillips GD, Leipold DD, Provenzano CA, Mai E, Johnson HA, Gunter B, Audette CA, Gupta M, Pinkas J, Tibbitts J (2012) The effect of different linkers on target cell catabolism and pharmacokinetics/pharmacodynamics of trastuzumab maytansinoid conjugates