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Description
Since there is no evidence that GSTM1 has superoxide dismutase nor catalase activity to metabolize O 2 - and H 2 O 2 , respectively, it is likely that the elevation in ROS levels in VSMCs after loss of GSTM1 indirectly results from accumulation of endogenously generated RAs that alter the redox state
Unravelling the epigenetic impact: Oxidative stress and its role in male infertility-associated sperm dysfunction
and J.Z
A novel two-nucleotide deletion in the ornithine transcarbamylase gene causing fatal hyperammonia in early pregnancy
