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However, this resistance can be mitigated by oxidized phosphatidylcholine (OxPC) produced during ferroptosis, which inhibits iNOS production by preventing NF-B binding to the iNOS promoter (Friedl et al

The interaction of cell death mechanism in PD Glial cells participate in neuronal death through inflammatory factors and metabolic regulation, and their cross-linking with cell death pathways further strengthens the pathological cascade reaction

, poly ADP ribose polymerase [PARP]), together leading to an increase in the NADH/NAD + ratio and promoting ROS production by mitochondrial respiratory complex I due to NADH overload, eventually resulting in reductive stress (38, 105, 107)

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