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Activation of miR-34a/SIRT1/p53 signaling contributes to cochlear hair cell apoptosis: implications for age-related hearing loss
The results validate the existence of oxidative stress in both liver and SNr across two murine models of HE induced by thioacetamide (TAA) and bile duct ligation (BDL)
The concentration of the main byproduct, DP1 [t R = 27.8 min], increased during the five hours of irradiation
Inactivation of -catenin, by contrast, results in dramatically reduced proliferation of hair progenitor cells and premature induction of the catagen (destructive) phase
