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UV exposure generates large amounts of reactive oxygen species in the skin, leading to oxidative damage of cellular components including DNA, proteins, and lipids
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DOI: 10.4103/sjg.sjg_480_22
Taken together, these findings suggest that APR-246 could induce tumor cell death through both reactivation of mutant p53 and inhibition of cellular thiol-dependent redox system proteins (GPX4, TXNDR1, GRX1), providing novel combinatorial strategies for AML therapy
