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It includes multifactorial or polygenic HTG (formerly familial HTG or type 4 hyperlipoproteinaemia with increased VLDL-TG concentration), dysbetalipoproteinaemia (formerly type 3 hyperlipoproteinaemia or dysbetalipoproteinaemia or remnant disease) with elevated concentration of VLDL remnants and chylomicron remnants as a result of their impaired catabolism, and combined hyperlipoproteinaemia (formerly type 2b hyperlipoproteinaemia or familial combined hyperlipoproteinaemia) with elevated VLDL-TG and LDL-C concentration [212]

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The mechanism of enzymic formation of homogentisate from p-hydroxyphenylpyruvate

LCHAD deficiency is an autosomal recessive trait and affected infants present in infancy with acute hypoketotic hypoglycemia