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The ion trapping theory demonstrates that the acidic pH of the gastric lumen facilitates passive cellular diffusion of the non-ionized, lipid soluble NSAIDs followed by re-conversion into ionized and lipophobic form within the neutral cytosolic pH of the gastric mucosal cell where they are assumed to induce subsequent toxicity [24]
Normally, cellular GSSG content is extremely low so that protein mixed disulfide formation is limited
Wound healing is a complicated process
Diversity and dialogue in immunity to helminths
