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Serum eotaxin-1 was also examined in the context of therapy response: in the 2022 vedolizumab-response study, higher baseline eotaxin-1 levels were observed in patients who went on to respond to vedolizumab, compared to non-responders (49)

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gasseri inhibited RSL3-induced ferroptosis in NCM-460 cells, with the mechanism involving activation of the GSH/GPX4 signaling pathway

Such models are used to evaluate receptor activation patterns, biomarker expression, and signaling feedback mechanisms under controlled laboratory conditions, with emphasis on reproducibility and mechanistic interpretation rather than applied outcomes