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Description
Elevated iron in the SNpc, reduced glutathione, and widespread oxidation of lipids document the oxidative milieu in post-mortem PD brains, whereas deficits in complex I activity and impaired mitophagy underscore the pivotal role of dysfunctional mitochondria in reactive oxygen species (ROS) overload

All reactions were performed in triplicate based on a standard curve method

10.1002/hep.21523 179 Serafini-CessiF

Preliminary studies for this group of animals have been reported (Israel et al., Experiment 2
