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(2018) 378:e34

It was hypothesized that in HepG2, miR-370 contributed to hepatic triglyceride accumulation by directly downregulating CPTIA and promoting lipogenesis through the activation of miR-122

showed that, in MCD-fed MASLD mice, the knock-in of cGPX4 relieved steatohepatitis and liver injury, whereas the knock-in of iGPX4 produced the opposite result

Curr Treat Options Neurol
