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they can be thrombogenic (provoke clot formation), so they are generally avoided in patients at risk for thrombotic events
28 HernndezI.ChacnO.RodriguezR.PortielesR.LpezY.PujolM.et al (2009)
This depletion is compounded by myeloid-derived suppressor cells (MDSCs) that secrete arginase-1 to catabolize arginine, another amino acid crucial for T cell activation and proliferation [42]
The mechanism is largely angiogenic: BPC-157 upregulates VEGF (vascular endothelial growth factor) and activates VEGFR2, stimulating the formation of new blood vessels in damaged tissue
