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Engineered bacteria manipulate cysteine metabolism to boost ferroptosis-based pancreatic ductal adenocarcinoma therapy

Bax protein is a proapoptotic member that acts by forming pores on the mitochondrial outer membrane, allowing cytochrome c to translocate into the cytoplasm, resulting in the loss of energy production and the activation of proteolytic cascade leading to apoptosis (Westphal et al., 2014)

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Additionally, ALDH2 enhances antioxidant defenses by activating SLC7A11, which promotes intracellular GSH synthesis and decreases free iron (Fe2+) levels, alleviating ferroptosis-mediated cell injury (125)