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The role of ER stress and UPR in NDDs The UPR is a critical cellular mechanism activated in response to the accumulation of misfolded or unfolded proteins in the ER
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This opposed the possibility that a generic response to D-HIS could explain the increased GSH level seen in the pGSH1-GAP1 strain with D-HIS
our desire to change the past and ease our growing pains, while knowing that would never be possible, even if we could travel back in time
