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It is initiated through two principal pathways: the extrinsic (death receptor-mediated) and intrinsic (mitochondrial) pathways, which converge on the activation of executioner caspases (Fig
10.1016/j.cellsig.2009.12.003 38 ClaytonC
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This occurs when the bone marrow suddenly starts producing platelets more effectively, along with a rebound effect where platelet production overshoots normal levels
