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The glutamate secretion is sufficient to activate HIF-1 through a paracrine mechanism and inhibits the xCT glutamate-cystine antiporter, causing cysteine depletion, which leads to oxidative self-inactivation of PHD2 and ultimately HIF-1 accumulation [134]
[DOI] [PubMed] [Google Scholar] Hollis, F
J.JohnsonL
Recently, histone H3K9bhb kept chromatin in an activated state (48), suggesting a close association between histone Kbhb and chromatin accessibility
