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Unlike Tf, which is used to functionalize drug delivery systems through receptor targeting, ferritin leverages its natural TfR1 affinity and its hollow protein architecture to directly encapsulate therapeutic agents [315]

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Its well-known adverse events, e.g., interstitial pulmonary fibrosis, seizures, liver veno-occlusive disease, or sinusoidal obstruction syndrome [96] seem to be caused by glutathionyl-tetrahydrothiophene, a metabolite that derives from the irreversible GSH conjugation [97,98]

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