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Briefly, IGF-I heterozygous mice were obtained by crossing transgenic mice, line 129SV and Igf1tm1Arge [31]

82 Developmental stage-specific depletion of both HDAC1 and HDAC2 in radial glial progenitors attenuated Neurog2 expression, resulting in the mispositioning of IPs on the ventricular surface

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According to the results of six randomized, double-blind, placebo-controlled clinical trials performed with Fragment 176-191, this peptide does not cause increases in IGF-1 levels, insulin resistance, impaired glucose tolerance, or any negative effect on carbohydrate metabolism [1]