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Genetic deletion of Sult2b1 impairs cholesterol sulfate synthesis and exacerbates pro-inflammatory polarization, whereas exogenous cholesterol sulfate reverses this effect by modulating NADPH/ROS levels and the AMPK/CREB pathway (250)
The reason for the contradictory results in the in vitro experiments was probably the difference in the severity of the oxidation stress
Plasma levels of GHK are estimated at around 200 ng/mL at age 20 and decline steadily with age, which coincides with the slowdown in collagen production and wound repair that characterizes aging skin
