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Stress responses in neurons result in the inhibition of the neuronal NO synthase (NOS) activity and as a result a reduction in NO production

Patients infected with HIV exhibit diminished levels of GSH in plasma, erythrocytes, lymphocytes, and peripheral blood mononuclear cells (PBMCs), which is largely attributed to the enhanced inflammation and ROS production caused by the virus, and this GSH deficiency has been correlated with the dysregulated Th1-to-Th2 shift in immune response [4,32]

1988 Sep;22(9):65665

The role of miRNAs, lncRNAs, and EVs induced by viral factors in microglial activation and inflammatory signaling The role of miRNAs in microglial activation The roles and mechanisms of specific microRNAsincluding miR-204, miR-124, miR-505, and miR-34ahave been previously integrated into discussions of HIV-1 Tat-mediated microglial activation