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The ability of cancer cells to prevent apoptosis via increased ROS formation requires increased antioxidant defense, such as increased transcription of genes encoding thioredoxin (TXN)- and glutathione (GSH)-dependent enzymes, as well as other detoxification and antioxidant enzymes (Fig
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Thalassemia bone disease: A 19-year longitudinal analysis
Studies show this tweak affects how fast cells grow and how well they repair themselves in petri dishes and animal models
