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The study has shown that exogenous H 2 S treatment can alleviate Hcy-induced endothelial dysfunction by inhibiting mitochondrial toxicity (101)
Theres also a genetic predisposition, as we observe this condition frequently runs in families
They can inhibit apoptosis by dampening the activity of apoptosis-associated caspases, thereby promoting cell recovery
10.1016/j.neuropharm.2011.07.008 166 WarburgO
