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Studies involving aged mice and clinical and translational medicine have shown promising results [1], linking Glynac supplementation to better mitochondrial function, reduced cellular senescence, and improved exercise capacity

In both theory and practice, given the beneficial effects obtained [11,29], the counteraction of free radical formation (along with any effect on NO-level or eNOS expression) and its beneficial effects in neurodegenerative diseases support each others effects

One of the major challenges encountered is the heterogeneity in study design, sample size, population characteristics, and dosage and duration of supplementation