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Dysregulated signalling pathways driving anticancer drug resistance
The ferroptosis inhibitor liproxstatin-1 has been shown to reverse iron accumulation, endothelial damage, and increased atherosclerosis in Jak2VF chimeric mice and VFEpoR mice, 539 indicating a potential therapeutic target for mitigating erythrocytic ferroptosis-mediated cardiovascular risk
This NO- and VEGFR2-dependent activity is the proposed engine behind the peptides angiogenic and cytoprotective effects, and it dovetails with the broader NO-system framework advanced by the originating research group
PMID: 176 HuNLiWHongYZengZZhangJWuXet al
